Are you aging faster than you would like due to telomere shortening?
Current research shows that telomere shortening contributes to cellular aging and susceptibility to metabolic disorders and chronic disease. According to scientific research, telomere shortening may contribute to aging and age-related conditions. Telomeres are segments of DNA found at the ends of chromosomes. They can be prematurely shortened when exposed to environmental chemicals, oxidation, inflammation, and stress. This leads to DNA damage and contributes to early cell death.
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Inflammation is associated with aging and telomere shortening. Telomeres become shorter after each cell division until, eventually, chromosomal DNA reaches a critical point at which the cell can no longer divide (known as the Hayflick limit). Losing this protective telomere “armor” renders DNA vulnerable to damage and may increase an individual’s risk for accelerated aging and associated health conditions. If telomeres are maintained, then termination of cell division (senescence) and programmed cell death (apoptosis) can be delayed.
The test analyzes your average telomere length in white blood cells compared to the normal reference range for your age-matched population.
Shorter telomeres have been associated with cardiovascular disease, inflammatory disorders, metabolic syndrome, diabetes, cognitive decline, and other chronic degenerative conditions associated with aging.